DLS https://ar.iub.edu.bd/handle/11348/487 2026-04-24T21:20:19Z 2026-04-24T21:20:19Z Chakrabarty, Rhiney https://ar.iub.edu.bd/handle/11348/1082 2026-04-15T05:47:45Z 2025-12-01T00:00:00Z

Internship at Mymensingh Medical College Hospital Chakrabarty, Rhiney This internship was undertaken as a partial requirement for the Bachelor of Science in Microbiology and was completed at Mymensingh Medical College Hospital, where I gained practical exposure to real-world clinical and laboratory environments. The primary objective of this internship was to bridge theoretical knowledge with hands-on experience by observing and participating in routine diagnostic procedures, laboratory operations, sample handling, and data interpretation under professional supervision. Throughout the internship, I had the opportunity to work closely with experienced medical technologists and faculty members, enabling me to understand the workflow of clinical microbiology, patient sample processing, infection detection methods, and laboratory safety practices. This experience not only broadened my academic learning but also contributed to the development of essential professional skills such as communication, discipline, teamwork, and ethical responsibilities in patient-centered environments. Overall, the internship at Mymensingh Medical College Hospital served as a valuable foundation for my future career in microbiology and the broader health sector.

2025-12-01T00:00:00Z Chakrabarty, Rhiney https://ar.iub.edu.bd/handle/11348/1081 2026-04-15T05:47:45Z 2025-12-01T00:00:00Z

Biofilm Mediated Complications in Hospital and Environmental Setting Chakrabarty, Rhiney Biofilms are complex microbial communities that attach firmly to surfaces and grow within a dense extra cellular matrix composed of polysaccharides, proteins, and nucleic acids. Their ability to anchor to medical devices, hospital surfaces, industrial pipelines, and food processing equipment makes them a major concern for both healthcare and industry. In hospital environments, biofilms act as persistent reservoirs of pathogenic microorganisms. They reduce the activity of antibiotics, protect harmful bacteria from the host immune system, and play a central role in long-lasting infections linked with catheters, prosthetic implants, ventilator tubes, and surgical instruments. These microbial structures also contaminate critical areas such as operating theatres, sinks, and water outlets, which increases the risk of hospital-acquired infections and elevates patient morbidity and mortality.

2025-12-01T00:00:00Z Faruque, Shah M. Nair, G. Balakrish https://ar.iub.edu.bd/handle/11348/509 2023-12-06T05:19:02Z 2020-10-25T00:00:00Z

Molecular Ecology of Toxigenic Vibrio cholerae Faruque, Shah M.; Nair, G. Balakrish Toxigenic Vibrio cholerae is the etiological agent of cholera, an acute dehydrating diarrhea that occurs in epidemic form in many developing countries. Although V. cholerae is a human pathogen, aquatic ecosystems are major habitats of Vibrio species, which includes both pathogenic and nonpathogenic strains that vary in their virulence gene content. V. cholerae belonging to the O1 and O139 serogroups is commonly known to carry a set of virulence genes necessary for pathogenesis in humans. Recent studies have indicated that virulence genes or their homologues are also dispersed among environmental strains of V. cholerae belonging to diverse serogroups, which appear to constitute an environmental reservoir of virulence genes. Although the definitive roles of the virulence-associated factors in the environment, and the environmental selection pressures for V. cholerae-carrying virulence genes or their homologues is not clear, the potential for origination of new epidemic strains from environmental progenitors seems real. It is likely that the aquatic environment harbors different virulence-associated genes scattered among environmental vibrio’s, which possess a lower virulence potential than the epidemic strains. The ecosystem comprising the aquatic environment, V. cholerae, genetic elements mediating gene transfer, and the mammalian host appears to support the clustering of critical virulence genes in a proper combination leading to the origination of new V. cholerae strains with epidemic potential.

2020-10-25T00:00:00Z